Critical POU domain residues confer Oct4 uniqueness in somatic cell reprogramming

Regulation of Oct4 in stem cells and neural crest cells - Patel - 2022 - Birth Defects Research - Wiley Online Library

A Late Transition in Somatic Cell Reprogramming Requires Regulators Distinct from the Pluripotency Network - ScienceDirect

Proteomic Analysis of Mouse Oocytes Identifies PRMT7 as a Reprogramming Factor that Replaces SOX2 in the Induction of Pluripotent Stem Cells

Critical POU domain residues confer Oct4 uniqueness in somatic cell reprogramming

Evolutionary origin of vertebrate OCT4/POU5 functions in supporting pluripotency

Changing POU dimerization preferences converts Oct6 into a pluripotency inducer

In silico identification of natural product inhibitors against Octamer-binding transcription factor 4 (Oct4) to impede the mechanism of glioma stem cells

Site-specific Disruption of the Oct4/Sox2 Protein Interaction Reveals Coordinated Mesendodermal Differentiation and the Epithelial-Mesenchymal Transition* - Journal of Biological Chemistry

A unique Oct4 interface is crucial for reprogramming to pluripotency

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