BRCA2 promotes DNA‐RNA hybrid resolution by DDX5 helicase at DNA

DDX5 resolves R-loops at DNA double-strand breaks to promote DNA repair and avoid chromosomal deletions. - Abstract - Europe PMC

BRCA2 promotes DNA‐RNA hybrid resolution by DDX5 helicase at DNA breaks to facilitate their repair‡

Sources, resolution and physiological relevance of R-loops and RNA–DNA hybrids

DDX5 resolves R-loops at DNA double-strand breaks to promote DNA repair and avoid chromosomal deletions. - Abstract - Europe PMC

R-loop resolution and formation in the cell. (A) Prevention and

DNA-RNA hybrids at DSBs interfere with repair by homologous recombination

R-Loops and Its Chro-Mates

DDX5 mRNA-targeting antisense oligonucleotide as a new promising therapeutic in combating castration-resistant prostate cancer: Molecular Therapy

Role of the DEAD-box RNA helicase DDX5 (p68) in cancer DNA repair, immune suppression, cancer metabolic control, virus infection promotion, and human microbiome (microbiota) negative influence

Role of the DEAD-box RNA helicase DDX5 (p68) in cancer DNA repair, immune suppression, cancer metabolic control, virus infection promotion, and human microbiome (microbiota) negative influence

Role of the DEAD-box RNA helicase DDX5 (p68) in cancer DNA repair, immune suppression, cancer metabolic control, virus infection promotion, and human microbiome (microbiota) negative influence

BRCA2 promotes DNA-RNA hybrid resolution at DNA breaks to facilitate homologous recombination

BRCA2 controls DNA:RNA hybrid level at DSBs by mediating RNase H2 recruitment

RNA binding protein DDX5 restricts RORγt+ Treg suppressor function to promote intestine inflammation

Genome-wide R-loop analysis defines unique roles for DDX5, XRN2, and PRMT5 in DNA/RNA hybrid resolution