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CD209L/L-SIGN and CD209/DC-SIGN Act as Receptors for SARS-CoV-2

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Last updated 31 julho 2024

CD209L/L-SIGN and CD209/DC-SIGN Act as Receptors for SARS-CoV-2

Smoking and SARS-CoV-2 Impair Dendritic Cells and Regulate DC-SIGN Expression in Tissues

CD209L (L-SIGN) is a receptor for severe acute respiratory syndrome coronavirus

CD209L/L-SIGN and CD209/DC-SIGN act as receptors for SARS-CoV-2 and are differentially expressed in lung and kidney epithelial and endothelial cells

Full article: Immunouniverse of SARS-CoV-2

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Mechanisms of SARS-CoV-2 entry into cells

Human Cell Receptors and Downstream Cascades: A Review of Molecular Aspects and Potential Therapeutic Targets in COVID-19, Archives of Pediatric Infectious Diseases

Mechanisms of SARS-CoV-2 entry into cells

DC-SIGN and DC-SIGNR Interact with the Glycoprotein of Marburg Virus and the S Protein of Severe Acute Respiratory Syndrome Coronavirus

DC/L-SIGN recognition of spike glycoprotein promotes SARS-CoV-2 trans-infection and can be inhibited by a glycomimetic antagonist

SARS CoV-2 spike protein variants exploit DC-SIGN/DC-SIGNR receptor for evolution and severity: an in-silico insight

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Frontiers The role of DC-SIGN as a trans-receptor in infection by MERS-CoV

CD209L/L-SIGN and CD209/DC-SIGN Act as Receptors for SARS-CoV-2

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