Herpes simplex virus enhances chemokine function through modulation of receptor trafficking and oligomerization

Por um escritor misterioso
Last updated 30 julho 2024
Herpes simplex virus enhances chemokine function through modulation of  receptor trafficking and oligomerization
Herpes simplex virus enhances chemokine function through modulation of  receptor trafficking and oligomerization
Macrophage migration inhibitory factor is a valid drug target at the intersection of herpes simplex virus 1 replication and Alzheimer's disease-relevant cellular pathology
Herpes simplex virus enhances chemokine function through modulation of  receptor trafficking and oligomerization
Viruses, Free Full-Text
Herpes simplex virus enhances chemokine function through modulation of  receptor trafficking and oligomerization
Herpes Simplex Virus 2 Counteracts Neurite Outgrowth Repulsion during Infection in a Nerve Growth Factor-Dependent Manner
Herpes simplex virus enhances chemokine function through modulation of  receptor trafficking and oligomerization
Frontiers Modulation of cellular signaling by herpesvirus-encoded G protein-coupled receptors
Herpes simplex virus enhances chemokine function through modulation of  receptor trafficking and oligomerization
Secreted herpes simplex virus-2 glycoprotein G alters thermal pain sensitivity by modifying NGF effects on TRPV1, Journal of Neuroinflammation
Herpes simplex virus enhances chemokine function through modulation of  receptor trafficking and oligomerization
Herpes simplex virus enhances chemokine function through modulation of receptor trafficking and oligomerization
Herpes simplex virus enhances chemokine function through modulation of  receptor trafficking and oligomerization
Herpes Simplex Virus 2 Counteracts Neurite Outgrowth Repulsion during Infection in a Nerve Growth Factor-Dependent Manner
Herpes simplex virus enhances chemokine function through modulation of  receptor trafficking and oligomerization
International Union of Basic and Clinical Pharmacology. LXXXIX. Update on the Extended Family of Chemokine Receptors and Introducing a New Nomenclature for Atypical Chemokine Receptors
Herpes simplex virus enhances chemokine function through modulation of  receptor trafficking and oligomerization
Frontiers Residue Mutations in Murine Herpesvirus 68 Immunomodulatory Protein M3 Reveal Specific Modulation of Chemokine Binding
Herpes simplex virus enhances chemokine function through modulation of  receptor trafficking and oligomerization
Therapeutic targeting of chemokine interactions in atherosclerosis
Herpes simplex virus enhances chemokine function through modulation of  receptor trafficking and oligomerization
Herpes Simplex Virus 2 Counteracts Neurite Outgrowth Repulsion during Infection in a Nerve Growth Factor-Dependent Manner

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